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The symptomatic carotid artery: on inflammatory more disorder?

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The mental specialist ISSN: 0028-2804 (printed version) ISSN: 1433-0407 (electronics version) Table OF CONTENTS Abstract volume 71 Issue 12 (2000) pp 955-962 overview : The unstable Karotisstenose - an inflammatory illness? The symptomatic carotid artery: on inflammatory more disorder? G. Stoll (1), S. Jander (1), M. Sitzer (4), W. Mueller (2), W. Sandmann (3), M. Siebler (1)   (1) neurological hospital of the Heinrich Heine university Duesseldorf (2) pathological Institut of the Heinrich Heine university Duesseldorf (3) hospital for container surgery and Nierentransplantation of the Heinrich Heine university Duesseldorf (4) neurological hospital of the Johann Wolfgang Goethe university Frankfurt   Summary Arterioarterielle Thrombembolien from extrakraniellen Stenosen of the A. carotid interna (ACI) represent a frequent cause of cerebral Ischaemien. The impact accumulation risk depends not exclusively on the Stenosegrad: High-grade (> 70%ige) ACI Stenosen has an annual impact accumulation risk of 13%, if they were symptomatic within 6 months, opposite only 1-2% with before asymptomatischen Stenosen. The Pathomechanismen, which makes an acute neurological illness in the sense of the Plaquedestabilisierung from a chronic arteriosklerotischen Karotisplaque, is to a large extent unclear. There are increasing references on the fact that inflammation processes are involved in the emergence of the Arteriosklerose generally and the Plaquedestablisierung in the special one. On the basis from container preparations to Karotisthrombendarteriektomien it could be shown that the extent of the t-cell and macro phage infiltration in the gefaesswand and the Expression of Matrixmetalloproteinasen with clinical and sonologischen indications of the Plaquedestabilisierung in the sense of Mikroembolien correlate. Thus new antiinflammatorische therapy beginnings possibly result in the case of symptomatic carotid processes. Summary Arterioarterial thromboembolism from extracranial internal carotid artery (ICA) stenosis is important on pathogenic mechanism OF ischemic stroke. However, even A of high degrees of ICA stenosis carries A greatly variable annual risk OF stroke, as high as 13% following A recent occurrence OF transiently or minor cerebral ischemia or as low as 1-2% in clinically asymptomatic patients. There is increasing evidence that inflammatory processes play A cent ral role in atherosclerosis and particularly into plaque destabilization converting chronic atherosclerosis into acute on neurological more disorder. In thromboendarterectomy specimens from patients with high degrees of ICA of stenoses, the extent OF inflammatory infiltration and the expression OF matrixmetalloproteinase-9 correlated tons clinical and ultrasonic feature OF plaque destabilization look for as cerebral microembolism. Inflammation might become A new therapeutic target into symptomatic carotid artery disease.   Keywords Karotisstenose · Mikroembolien · T-cells · Tissue factor · Matrixmetalloproteinase Keywords Carotid artery stenosis · Microemboli · T cells · Tissue factor · Matrixmetalloproteinase Article in pdf format (328 KB) On-line one publication: November 24, 2000 LEFT Helpdesk © Springer publishing house Berlin Heidelberg 2000

 

 

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